"The Whole Elephant Institute: the Article that triggered CCP's

Transnational Repression Crime Against Dr. Tongwen Wang"

《全象学院：引發中共跨國鎮壓王彤文博士的文章》

 

Transnational Repression by CCP is a Crime:

中共跨国镇压是犯罪：

Transnational Repression

Persecution in America:

中共渗透美国對大法修煉者的迫害：

https://www.thewholeelephant.info/persecution-in-america

https://www.thewholeelephant.info/save-sentient-beings

https://www.thewholeelephant.info/flushing-hate-crimes

Dr. Lotus King Weiss' FBI report on CCP's Transnational Repression through its infiltrates:

李天水博士向聯邦調查局匯報中共跨國鎮壓案：

https://youtu.be/d8IzBQqqZMI

THE ARTICLE That Triggered CCP's Transnational Repression towards Dr. Tongwen Wang:

下面是引發中共跨國鎮壓王彤文博士的文章：

"A Wake-Up Call from the Microcosm"

《從微观世界傳來的警钟》

PureInsight | May 20, 2002

發表在《正見網》二零零二年五月二十日

Author:

Dr. Tongwen Wang

Principal Investigator in Cancer and Autoimmune Diseases

Department of Immunology

Virginia Mason Research Center

University of Washington

作者：

王彤文博士

癌症和自身免疫性疾病的首席研究员

免疫学系

弗吉尼亚梅森研究中心

华盛顿大学

One early spring day, I was on my way to work. A stranger greeted me and asked me what I did for a living. After learning that I am a cancer researcher, his eyes lit up and asked: “Are you going to find a cure for cancer?” His question plunged me deep into thought and now I would like to share some ideas with you.

早春的一天，我正在上班的路上。一个陌生人向我打招呼，问我做什么工作。當得知我是癌症研究人员后，他眼睛一亮，问道: “你要找到治愈癌症的方法吗？”他的问题让我陷入了深思，现在我想与您分享一些想法。

Ever since my grandmother passed away so suddenly from malignant liver cancer about twenty years ago, the word “cancer” has never left my mind and heart.

It was the innocent hope from my young heart that one day I would find a cure that has led me to this stage of my career.

Since 1988, I have followed the path of the reductionism-based modern science approach to study Biology at multiple levels: from Anatomy to Histology, Cell Biology, and, finally, to Molecular Biology, using yeast as a model system to study gene regulation.

After I obtained my Ph.D. degree, I felt I was ready to go face-to-face with cancer and therefore stepped into the field of cancer research.

I do research in this field, like many others, via observing cells in artificial systems, specifically, what we call in vitro systems, which are cell lines grown in petri dishes.

自从大约二十年前我的祖母突然死于恶性肝癌, “癌症”这个词就从未离开过我的心。

就是因為年轻時心怀着天真无邪的希望，希望有一天我能找到一种治愈癌症的方法，将我带到了我职业生涯的这个阶段。

自 1988 年以来，我沿着基于还原论的现代科学方法在多个层面研究生物学：从解剖学到组织学、细胞生物学，最后到分子生物学，使用酵母作为模型系统来研究基因调控。

在我获得博士学位之后，我觉得自己已经准备好挑战癌症，因此踏入了癌症研究领域。

我在这个领域做研究，像许多其他人一样，通过观察人工系统中的细胞，特别是我们所说的体外系统，这是在培养皿中生长的细胞系统。

In 1992, when I started my postdoctoral training at Massachusetts General Hospital in Boston, I “met” this powerful protein called Transforming Growth Factor-beta (TGF-beta). This molecule is a potent suppressor of cell growth.

1992 年，当我开始在波士顿马萨诸塞州总医院进行博士后培训时，我“遇到”了一种名为转化生长因子-β (TGF-β) 的强大蛋白质。该分子是细胞生长的有效抑制剂。

In our body, there is a large number of TGF-beta-like proteins, all of them are very powerful, in that each of them is in charge of the formation and maintenance of different major organs of our body. Most of these proteins are potent inhibitors of cell growth.

在我们的身体里，有大量的TGF-β样蛋白，它们都非常强大，每一个都负责我们身体不同主要器官的形成和维护。大多数这些蛋白质是细胞生长的有效抑制剂。

In our body, there is also a large group of proteins whose functions are to actively promote cell growth.

在我们的身体中，还有一大群蛋白质，其功能是积极促进细胞生长。

Here the ancient wisdom of China, the theory of the balance of Yin and Yang in the macrocosm, is beautifully reflected at the molecular level, in the microcosm of the cell.

在这里，中国古老的智慧，宏观阴阳平衡的理论，在分子水平上，在细胞的微观上得到了很好的体现。

Research conducted over the past twenty years has led to the elucidation of the detailed molecular network in a normal cell which goes through a highly regulated life cycle of growth, specialization (we call differentiation), aging and death.

过去二十年进行的研究已经阐明了正常细胞中详细的分子网络，展現了正常细胞會经历有高度调节機制的生长、專業化（我们称为分化）、衰老和死亡的生命周期。

In every step of the life of a cell, we can “hear the song” and “see the dance” of the interplay of the Yin and Yang factors in great harmony. The disruption of the balance between these two factors is recognized as the foundation for uncontrolled cell behaviors, one of which manifests as cancer.

在细胞生命的每一步中，我们都可以“听歌”、“看舞”，觀察列陰陽之間和谐地在相互作用。陰陽之间平衡的破坏被认为是細胞失控的基础，其中之一表现为癌症。

How does a normal cell turn into a cancer? What went wrong?

正常细胞是如何变成癌细胞的？什么地方出了错？

A normal cell responds to environmental cues to determine when it can enter the growth phase, called the cell cycle, which consists of several distinct stages, named G1, S, G2 and M. There are gates between each stage. The cell has to meet certain requirements before it can move on. These gates are very important, since if there is something wrong in the cell, the gates will serve to safeguard the cell by stopping it at that phase until the problem is somehow fixed.

正常细胞对环境线索作出反应，以确定何时可以进入生长阶段，称为细胞周期，它由几个不同的阶段组成，称为 G1、S、G2 和 M。每个阶段之间都有门。细胞必须满足某些要求才能继续前进。这些门非常重要，因为如果細胞中出现问题，门将通过在该阶段中停止来保护細胞，直到问题以某种方式得到解决。

If the problem cannot be fixed, the cell would trigger an alarm system that, amazingly, leads to a well-orchestrated death program.

如果问题无法解决，细胞会触发警报系统，令人惊讶的是，它会导致精心策划的死亡程序。

Thus, a normal cell acts in accord with the system it belongs to. When mistakes occur, the cell has a mechanism to “sacrifice” itself for the benefit of the whole.

因此，一个正常的细胞按照它所属的系统行动。当错误发生时，细胞有一种机制，为了整体的利益而“牺牲”自己。

Contrariwise, a cancer cell somehow outsmarts the laws at each gate of the check points between the different phases of growth, thereby continuing to grow in number. The death mechanism is also abolished so that they reach “immortality”. Of course, such temporary immortality is followed by the death of the whole, which reflects, interestingly, a very ignorant but totally selfish “spirit”.

相反，癌细胞以某种方式在不同生长阶段之间的检查点的每个门上聪明地超越了法律，从而在数量上继续增长。死亡机制也被废除，使他们达到“不朽”。当然，这种暂时的不朽伴随着整体的死亡，有趣的是，这反映了一种非常无知但完全自私的“精神”。

In the past twenty years, cancer researchers have learned that it takes many steps for a cell to accumulate the various protein mistakes that eventually wipe out all major safe-guarding mechanisms at the level of a cell.

在过去的 20 年里，癌症研究人员了解到，细胞需要经过许多步骤才能积累各种蛋白质错误，最终消除细胞水平上的所有主要安全保护机制。

Then the cancer cells start to metastasize, during which it again violates multiple system laws, including the laws of the immune system.

然后癌细胞开始转移，在此期间它再次违反多个系统法則，包括免疫系统法則。

Like the policeman, the immune system normally provides constant surveys of the body systems to eliminate abnormal cells.

像警察一样，免疫系统通常会持续检查身体系统以消除异常细胞。

The real mystery is why the cancer cells can manage to accumulate so many mistakes without being eliminated.

真正的谜团是为什么癌细胞可以设法累积这么多错误而不被消除。

Within the microcosm of a cell, we know that many safeguarding mechanisms are in place. Within the “midcosm” of a human body, we know that there are also many safeguarding mechanisms in place. Why does every safeguarding mechanism fail in a cancer patient?

在細胞這個微觀"小宇宙"中，我们知道存在许多保护机制。在人體這個"中宇宙"內，我们知道也有许多保护机制。那么为什么癌症患者的所有保护机制都会失效？

Some biologists believe that cancer is due to some mistakes at the gene level, which allow the genetic material to be unstable, called genetic instability, thus leading to large scale mistakes at the gene level.

一些生物学家认为，癌症是由于基因水平上的一些错误导致遗传物质不稳定，称为遗传不稳定性，从而导致基因水平上的大规模错误。

However, a normal cell knows how to fix a mistake at the gene level and also knows how to initiate the death program when it fails to fix the problem.

然而，正常细胞知道如何在基因水平上修复错误，也知道如何在无法修复问题时启动死亡程序。

Many labs, including my own, study how cells communicate with each other via proteins (www.vmresearch.org; “laboratory research”). TGF-beta is made by almost every cell in our body. When it is released outside of a cell, TGF-beta serves as a signal to “instruct” surrounding cells that have a unique set of proteins that can recognize and bind to it. Once these binding proteins, “receptors”, which are sitting on the cell surface, bind to TGF-beta, these receptors will then “talk” to proteins inside of the cell. The detailed steps of protein-protein communication inside of a cell in response to an outside-of-cell protein are mapped out carefully by many labs in the signal transduction research community.

许多实验室，包括我自己的，研究细胞如何通过蛋白质相互交流。 TGF-β 几乎由我们体内的每个细胞产生。当他被释放到细胞外时，TGF-β 作为信号“指令”周围细胞，这些细胞具有一组独特的蛋白质，可以识别并结合他。一旦这些位于细胞表面的结合蛋白“受体”与 TGF-β 结合，这些受体就会与细胞内的蛋白质“对话”。信号傳導研究界的许多实验室都仔细绘制了细胞内蛋白质與蛋白质之間通讯以响应细胞外蛋白质的详细步骤。

After six years of intensive and expensive studies, we have now identified an interesting functional mechanism for TGF-beta.

经过六年的密集和昂贵的研究，我们现在已经确定了一个有趣的 TGF-β 功能机制。

It is known to everyone in this field that there are a group of proteins inside of the cell, “Smad”, that are critical for carrying out the instructions of TGF-beta to suppress cell growth. In fact, many cancers, such as colon cancer, pancreatic cancer, and head and neck cancer, are all associated with defects in these Smad proteins. Only recently have we found that Smad carries out their mission through directly talking to an extremely important protein system inside the cell.

该领域的每个人都知道，细胞内有一组蛋白质“Smad”, 它们对于执行 TGF-β 抑制细胞生长的指令至关重要。事实上，许多癌症，如结肠癌、胰腺癌和头颈癌，都与这些 Smad 蛋白的缺陷有关。直到最近，我们才发现 Smad 通过直接与细胞内一个极其重要的蛋白质系统对话来执行他们的任务。

This protein system consists of a large number of proteins that all work together to do the following jobs: 1) mark old, aged, or dysfunctional proteins for destruction; and 2) help almost every aspect of cellular function via orderly breaking down proteins to fine-tune the levels of each protein that works as a regulator in the cell. This system also is essential for the immune system to find what is wrong when virus and bacteria enter the body, or when a cell behaves abnormally. This protein system is called “the proteasome system”. The malfunction of this system also blocks the function of TGF-beta as a suppressor of cell growth.

这个蛋白质系统由大量蛋白质组成，他們共同配合完成以下工作：

1）标记舊的、老化的或功能失调的蛋白质以进行降解；

2）通过有序分解蛋白质来微调作为细胞生長调节因子的每种蛋白质的水平，从而帮助细胞功能的几乎每个方面。

3）当病毒和细菌进入人体或细胞表现异常时，该系统对于免疫系统发现问题也是必不可少的。

这种蛋白质系统被称为“蛋白質降解器”。该系统的故障也會阻止 TGF-β 作为细胞生长抑制因子的功能。

When I was pondering the meaning of this most recent finding in my lab, my friend Dr. Lili Feng called me one day. Lili is an Associate Professor at Baylor College of Medicine. Both Lili and I practice Falun Dafa, an ancient mind-body practice now broadly known to the public largely due to the recent persecution of Falun Dafa in China (www.falundafa.org). I knew Lili was carrying out a project to examine the effect of practicing Falun Dafa on cells of the immune system.

当我在思考这个自己實驗室中最新发现的意义时，有一天，我的朋友封莉莉博士给我打电话。莉莉是贝勒医学院的副教授。莉莉和我都修炼法轮大法，这是一种古老的身心修炼，现在广为人知，主要是由于最近法轮大法在中国的迫害（www.falundafa.org）。我知道莉莉正在做一个项目，研究修炼法轮大法对免疫系统细胞的影响。

Lili told me that she has completed her studies on comparing the level of 12,000 genes in Falun Dafa practitioners and non-practitioners. To my great surprise, she mentioned some genes in the proteasome system. I asked her to send me the original data and decided to take a careful look. From that point on, an amazing stream of enlightening information flowed into my research system.

莉莉告诉我，她已经完成了比较法轮大法修炼者和非修炼者多核白血球細胞中12000个基因水平的研究。令我惊讶的是，她提到了蛋白質降解器Proteasome中的一些基因。于是我让她把原始数据发给我，决定仔细看看。从那时起，一股惊人的启发性信息流入了我的研究系统。

The data Lili sent to me was a pile of numbers assembled randomly from the experiments. But from the pile of the numbers, one clear image came out: the gene expression level corresponding to more than 10 different proteins in the proteasome system are drastically down-regulated in Falun Dafa practitioners’ immune cells.

莉莉发给我的数据是一堆实验随机組合出来的数字。但从這一堆数字中，一个清晰的图像浮出水面：對應於蛋白質降解器Proteasome中有10多种不同的蛋白质的基因表達在法轮大法修炼者的免疫细胞中被大幅下调。

This would indicate that the proteasome system is down-sized. It would not make much sense if only this system is down-sized, since the lack of sufficient proteasome system would lead to the accumulation of junk and old proteins.

这将表明蛋白質降解器proteasome体系统減少了。如果只是減少这个系统的规模，那麼就會有大問題，因为缺乏足够的蛋白質降解系统会导致垃圾蛋白和旧蛋白质的堆積。

Interestingly, in the same set of data, gene expression corresponding to more than 10 different proteins that belong to another protein system called “ribosome” are also drastically reduced. Ribosomes are responsible for making new proteins. I suddenly realized that the data is suggesting the coordinated down-sizing of the entire pipe-line of proteins production and protein consumption.

有趣的是：在同一组数据中，属于另一种称为“核糖体”的蛋白质系统的 10 多种不同蛋白质相对应的基因表达也大幅减少。核糖体负责制造新的蛋白质。我突然意识到，这组数据表明细胞中整个蛋白质生产和蛋白质消耗管道的规模正在协调性地同时缩小。

Lili then mentioned to me that she has read papers regarding correlations between proteasome system size and activity with longevity, in mouse experiments. Dr. Allen Taylor from Boston University reported that when the food supply was restricted, mice live longer and their proteasome system is down-sized (ref 1-3). I then found a paper that reported the correlation of increased proteasome system activity with many different diseases. In this paper, it was reported that the highest proteasome system activity was found in cancer cells (ref 4). A third paper from Lili added the final touch to an idea that started to surface (see below). In this paper (ref 5) it is reported that, from careful studies of protein metabolism in a cell, it seems that 1/3 of new proteins are immediately destroyed after they are made. Thus, even "normal cells" are working in a very busy and wasteful state.

莉莉随后向我提到，她曾阅读过有关蛋白质降解器系统Proteasome System大小和活性与寿命之间相关性的论文，是在小鼠实验中展现出来。波士顿大学的 Allen Taylor 博士报告说，当食物供应受到限制时，小鼠的寿命会更长，它们的蛋白质降解器系统也会缩小（参考文献 1-3）。然后我发现了一篇论文，报告了蛋白质降解器系统Proteasome System之活性增加与许多不同疾病的相关性。在这篇论文中，据报道，在癌细胞中发现了最高的蛋白质降解器系统Proteasome System之活性（参考文献 4）。 莉莉寄给我的第三篇论文为一个开始浮出水面的想法添加了最后的润色（见下文）。在这篇论文（参考文献 5）中，据报道，从对不般所谓正常细胞中蛋白质代谢的仔细研究来看，似乎有 1/3 的新蛋白质在制造后立即被破坏。因此可见，即使是我们认为的正常细胞也在非常忙碌和浪费的状态。

Lili and I started to send emails back and forth. Lili has a wonderful sense of humor and a wild imagination. One day she asked me, “Do you know what the proteasome is in the microcosm?” Then she answered for me, “The black hole.” Then she sent me a set of reports on how active the black holes are now in the Universe. “You see”, she said, “The proteasomes are very busy when our cells are sick, and what does it mean when the black holes in the macrocosm of the universe we live in are very busy?” (She was hinting that our universe is also sick!)

When I heard that, I thought of the phenomenon of our modern lifestyle: mass production and mass consumption.

Isn’t it amazing that the different cosmic systems of cell, body, society and the entire Universe, from micro to macro, exhibit such striking similarities and correspondence?

莉莉和我开始来回发送电子邮件。莉莉有着绝妙的幽默感和天马行空的想象力。有一天她问我: “你知道在微观宇宙中，蛋白质降解器其实是什么吗？” 然后她替我回答: “黑洞。”然后她给我发了一组关于黑洞现在宏观宇宙中有多活跃的报告。 “你看，”她说, “细胞生病时蛋白质降解器非常忙碌，而当宏观宇宙中的黑洞非常忙碌时，这意味着什么？” （她是说：人类生存的宇宙也生病了！）

听到这里，我想到了我们现代生活方式的现象：无度生产和无度消费。

细胞、人体、人类社会和整个宇宙的不同系统，从微观到宏观，表现出如此惊人的相似性和对应性，是不是很神奇？

At this point, when I went back to think about the question, “What makes the cancer cells accumulate so many mistakes and allow it to violate so many different safeguarding mechanisms,” a simple but clear answer came to my mind, “It is the hyper-metabolism rate of the proteins!” If all cells in a body are in the state of mass-production, the proteasome system is likely to be overloaded and unable to break down old and broken proteins, which then carry out wrong things, which further disrupt the balance. Since the proteins are the real players in all of the functions of a cell, when bad proteins cannot be eliminated, they will continue to do bad things till the entire system is out of control. No matter how hard the cell tries to increase the amount of proteasome production, if the metabolism continues to increase, the cell will eventually fail to manage. The increased proteasome level seen in cancer cells likely reflects such a last struggle the cell trying to regain the balance.

说到这里，当我回过头去思考“是什么让癌细胞积累了这么多错误并让它违反了这么多不同的保护机制”这个问题时，我想到了一个简单而明确的答案：

蛋白质的高代谢率！

如果体内所有细胞都处于大量无度之生产状态，那么蛋白质降解器系统 Proteasome System 很可能会超载，无法分解旧的和损怕了的蛋白质，从而进行错误的操作，从而进一步破坏平衡。由于蛋白质是细胞所有功能的真正参与者，当坏蛋白质无法消除时，它们会继续做坏事，直到整个系统失控。无论细胞如何努力增加蛋白质降解器Proteasome的数量，如果新陈代谢继续增加，细胞最终将无法管理。在癌细胞中观察到的蛋白质降解器Proteasome水平升高可能反映了细胞试图重新获得平衡的最后挣扎。

I cannot help wondering how many of the diseases modern people are experiencing are the result of the hectic lifestyle they have, the mental stress they are under, and the endless pursuits in which their minds and hearts engage. All these can, through the unique human psycho-neuroendocrine system, transmit to the cell level orders to increase the cellular metabolism, which, when it overwhelms the proteasome system, leads to the accumulation of cellular mistakes, and, finally, to the downfall of the body system. To take a step further, is the endless desire for more money, more goods, more recognition and more power also closely linked to many of society’s diseases?

我不禁想知道现代人所患的疾病有多少是他们忙碌的生活方式、他们承受的精神压力以及他们的思想和心灵无休止的追求的结果。所有这些都可以通过人体独特的精神神经内分泌系统，将指令传递到细胞层面，增加细胞之新陈代谢，当这种增加压垮了蛋白质降解系统之承受力时，必将导致细胞中蛋白质错误的堆积，最终导致身体整个系统的垮台。更进一步去思考，那么人类对更多金钱、更多商品、更多认可和更多权力的无休止的渴望是否也与许多当今种种人类社会层面之疾病密切相关呢？

Then what is the cure for cancer? What is the cure for all of society’s problems? What can slow down the activities of the black hole in the Universe? The problem of curing cancer is as gigantic as the latter two problems. But is there a common cause for all three? Is there a Universal Law, violation of which will lead to the manifestation of all cosmic problems, from small to big? Is it possible that everything we can see in this physical world, is a mere manifestation of a something we vaguely call consciousness?

那么治疗癌症的方法是什么呢？

那么解决所有人类社会问题的方法是什么呢？

那么可以减缓宇宙中黑洞的活动之的方法又是什么呢？

这样看来，治愈癌症的问题与后两个问题一样的巨大!

但是，这三种层次的疾病是否有着共同的原因?

有没有一个宇宙法则，违反他会导致所有宇宙问题从小到大的表现？

有没有可能我们在这个物理世界中看到的一切，都只是一种我们模糊地称之为"精神"这东西的表现？

Here comes this forbidden gray zone for modern science: the realm of spirit and consciousness.

Modern science locks it outside and believes that we can only understand nature by cleanly separating out matter from spirit.

But what is the nature of spirit?

What is its connection with matter?

Without knowing the answers for these questions, can we truly be so comfortable and so confident that we can understand the laws of our body and the laws of this Universe at this physical level?

此念让我们进入了一个现代科学的禁区之灰色地带：

精神和意识的领域。

现代科学把他们锁在外面，并认为只有将物质与精神彻底分开，我们才能理解自然。

但精神的本质是什么？

它与物质有什么联系？

在不知道这些问题的答案的情况下，我们真的能如此自在、如此自信，以至于我们可以在这个物理层面上去全面了解我们身体的规律和这个宇宙的规律吗？

Prior to my trip to Boston, I was reading a new issue of Science, which had on its cover a famous painting by Michelangelo Merisi da Caravaggio (1573-1610), of the Greek myth of Narcissus. Narcissus, upon looking into the water, saw his own reflection, and then fell deeply in love with his own image. He could not let go of his fascination with this image but, instead, poured in all of his attention, let it consume all of his energy, and finally, he died.

在我去波士顿之前，我正在阅读新一期的《科学》杂志，其封面是米开朗基罗·梅里西·达·卡拉瓦乔（Michelangelo Merisi da Caravaggio，1573-1610 年）描绘的希腊神话中关于美男子纳西索斯变成水仙花的名画。纳西索斯在水中看到了自己的倒影，便深深地爱上了自己的形象。他无法放下对这幅画面的迷恋，反而倾注了所有的注意力，让它消耗了他所有的能量，最后，他死了。

At the very beginning, I thought, “Why couldn’t Narcissus tell that that was his own image?” Of course, at that time, I guess, they did not have mirrors. But then I asked, “Why didn’t he take a careful look at himself? If he had done that, he would have found out that there were lots of similarities between his own hands and clothes and those in the image”.

一开始，我想: “为什么那喀索斯不知道那是他自己的形象呢？”当然，那个时候，我猜，他们没有镜子。但后来我问: “他为什么不仔细看看自己？如果他这样做了，他会发现自己的手和衣服与图像中的有很多相似之处。”

Then I smiled, “Actually, how many of us remember to take a look at ourselves in our lives? When we have problems, we look at everything outside, except ourselves. Birth, disease, aging and death, we all look for answers from outside. We have spent so much of our resources to find the cures for diseases. We are now hoping that someday there will be a super-computer that will enlighten us with respect to the mysteries of life. But what if this entire physical world is a delusional world like what has been taught since a long time ago by ancient sages?

然后我笑道: “其实，我们有多少人记得在生活中审视自己？当我们遇到问题时，我们会看外面的一切，除了我们自己。生、病、老、死，我们都在外面寻找答案。我们花费了大量资源来寻找治疗疾病的方法。我们现在希望有一天会出现一台超级计算机，它会启发我们了解生命的奥秘。但是，如果这整个物质世界是一个像很久以前古圣所教导的那样的迷的世界呢？”

Have we seen enough correspondence between the Universe, the human society, the human body and the cell?

Is it possible that we have stared into our own shadow image for too long?

Is it time to find our true self and go home?

我们是否看到了宇宙、人类社会、人体和细胞之间的足够对应？

是否我们凝视自己的倒影已太久长？

是否，今天是找到真正的自己并回归真正的家园的时候了?

(Notes: After my talk, several Falun Dafa practitioners came to me and told me their personal experiences of how they or other practitioners recovered from various “incurable” diseases such as systemic sclerosis and various cancers through their cultivation practice.)

（注：我讲完之后，有几位法轮大法修炼者来找我，告诉我他们或其他法轮功学员通过修炼从系统性硬化症、各种癌症等各种“不治之症”中康复的亲身经历。）

References:

1. Scrofano MM, Jahngen-Hodge J, Nowell TR Jr, Gong X, Smith DE, Perrone G, Asmundsson G, Dallal G, Gindlesky B, Mura CV, Taylor A. The effects of aging and calorie restriction on plasma nutrient levels in male and female Emory mice. Mech Ageing Dev. 1998 Sep 15;105(1-2):31-44.

2. Scrofano MM, Shang F, Nowell TR Jr, Gong X, Smith DE, Kelliher M, Dunning J, Mura CV, Taylor A. Calorie restriction, stress and the ubiquitin-dependent pathway in mouse livers. Mech Ageing Dev. 1998 Nov 16;105(3):273-90.

3. Scrofano MM, Shang F, Nowell TR Jr, Gong X, Smith DE, Kelliher M, Dunning J, Mura CV, Taylor A. Aging, calorie restriction and ubiquitin-dependent proteolysis in the livers of Emory mice. Mech Ageing Dev. 1998 Apr 1;101(3):277-96.

4. Dutaud D, Aubry L, Henry L, Levieux D, Hendi KB, Kuehn L, Bureau JP, and Ouali A. Development and evaluation of a sandwich ELISA for quantification of the 20S proteasome in human plasma. J. of Immuno. Meth. 2002; 260:183-193.

5. Yewdell JW. Not such a dismal science: the economics of protein synthesis, folding, degradation and antigen processing. Trends in Cell Bio. 2001; 11 (7):294-297.

参考資料：

1. Scrofano MM、Jahngen-Hodge J、Nowell TR Jr、Gong X、Smith DE、Perrone G、Asmundsson G、Dallal G、Gindlesky B、Mura CV、Taylor A。衰老和卡路里限制对血浆营养水平的影响在雄性和雌性埃默里小鼠上的研究。《衰老机制开发杂志》1998 年 9 月 15 日；105(1-2):31-44。

2. Scrofano MM、Shang F、Nowell TR Jr、Gong X、Smith DE、Kelliher M、Dunning J、Mura CV、Taylor A. 小鼠肝脏中热量限制、压力和泛素依赖性途径。《衰老机制开发杂志》1998 年 11 月 16 日；105(3):273-90。

3. Scrofano MM、Shang F、Nowell TR Jr、Gong X、Smith DE、Kelliher M、Dunning J、Mura CV、Taylor A. Emory 小鼠肝脏中的衰老、热量限制和泛素依赖性蛋白水解。《衰老机制开发杂志》1998 年 4 月 1 日；101(3)：277-96。

4. Dutaud D、Aubry L、Henry L、Levieux D、Hendi KB、Kuehn L、Bureau JP 和 Ouali A. 用于定量人血浆 20S 蛋白酶体的夹心 ELISA 的开发和评估。《免疫学方法学杂志》2002; 260:183-193。

5. Yewdell JW。这不是一门令人沮丧的科学：蛋白质合成、折叠、降解和抗原加工的经济学。《细胞生物的趋势》2001年； 11 (7):294-297。